Overview
what it is and why it matters
Stress fractures occur when repetitive submaximal loading causes fatigue failure of bone at a rate exceeding the bone's ability to remodel. They are divided into low-risk fractures (tibia posteromedial, metatarsal shaft, fibula, iliac crest) that reliably heal with activity modification; and high-risk fractures (anterior tibial cortex, navicular, fifth metatarsal Jones zone, femoral neck, medial malleolus, patella, sesamoids) that have high nonunion or displacement risk and often require surgical fixation, particularly in athletes.
Female Athlete Triad (disordered eating, amenorrhea, osteoporosis) substantially increases stress fracture risk and should be screened in women with recurrent stress fractures.
Diagnosis
exam first, imaging secondPlain X-rays are insensitive early (2–3 weeks). MRI is the most sensitive test and defines fracture grading (Fredericson MRI grading I–IV). Bone scan is an alternative. CT is used for high-risk sites to assess cortical breach and displacement. DEXA scan for bone density in recurrent or atypical presentations.
Treatment Path
how care progresses at OSIActivity modification
Low-risk fractures: activity restriction from high-impact loading, low-impact cross-training.
Non-weight-bearing immobilization
For higher-grade low-risk fractures and initial management of high-risk fractures.
Bone health evaluation and treatment
Calcium, vitamin D; workup for metabolic bone disease.
Surgical Options at OSI
if non-operative care isn't enoughHigh-risk stress fractures (especially navicular, femoral neck tension-side, anterior tibia "dreaded black line") and fractures with cortical breakthrough are managed operatively in active patients.
Further Reading
authoritative sourcesExternal patient-education references and related OSI pages for additional background: